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The ubiquitous zinc-binding ADA1 is the isoform whose deficiency is associated with severe combined immunodeficiency (SCID)
There are several potential explanations for these observations, including (1) coupling with different G subunits may lead to the activation of protein kinase C (PKC) and ultimately an increase in -cell mass, especially under conditions of chronic administration, (2) their role in promoting protection against necrotizing or apoptotic -cell death 76,77 , and (3) activation of phosphatidylinositol 3 kinase y-subunit (PI3K), a kinase identified as a component of the neuropeptide Y signaling cascade which may aid in the correct localization of insulin granules to facilitate insulin secretion 78,79
Regulatory mechanisms controlling gene expression mediated by the antioxidant response element
Khunti et al
Hess DT, Matsumoto A, Kim SO, Marshall HE, Stamler JS
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